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This methyl transfer is critical to reactions involving proteins, phospholipids, DNA, RNA, creatine, hormones, development of cell membranes, degradation of histamine, and formation of norepinephrine and dopamine
Considering the rather subtle changes in the gut microbiota and the absence of significant alterations in SCFAs levels, we redirected our focus from microbial mediation towards elucidating the potential mechanism through which HT administration directly mitigates DQ-induced oxidative stress damage in the intestine
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