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Its primary mechanism is angiogenesis: BPC-157 triggers new blood vessel growth (via VEGF upregulation) in injured tissue, which accelerates tendon, ligament, gut, and joint repair

Beyond that, the evidence is per-compound plus the CagriSema (cag + sema) Phase 3 data as the closest human analog

Enteric glia regulate intestinal barrier function and inflammation via release of S-nitrosoglutathione

Dual Incretin Because both compounds reduce appetite and bodyweight, it is easy to assume they share a mechanism

12 Furthermore, rats treated with D-galactosamine to induce ALF show significant increase of Evans blue extravasation at coma