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And it doesn't raise calcium levels in a good way
These mechanisms collectively suggest that modifying population-wide or individual-level demographics in the microflora through live microorganisms/probiotics, fermentable substrates/prebiotics/synbiotics, combination derivations/postbiotics/dietary exposures/faecal microbiota transplantation (FMT) might exert influence on overall BC risk/treatment sensibilization-tolerability/quality-of life improvement
Presented for research literature review only Enache TA et al
In patients with MCAS, ongoing mediator release (histamine, prostaglandins, leukotrienes, cytokines) can drive: Visceral hypersensitivity Chronic neuropathic-type pain Heightened itch signaling Sleep disruption Central amplification of sensory input Even when mast cell activity is partially controlled, neural sensitization may persist
Both carry copper ions that drive their biological activity
