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1 Introduction Sepsis-associated encephalopathy (SAE) represents a severe complication of sepsis, defined by diffuse brain dysfunction without direct central nervous system (CNS) infection ( Oxidative stress is central to SAE pathogenesis, driven by an imbalance between excessive reactive oxygen species (ROS) production and impaired antioxidant defense systems ( The antioxidant defense system, especially GSH metabolism, is linked to neurodegenerative diseases ( Proteomics and metabolomics enable systematic profiling of protein expression and metabolic signatures

Curr Opin Plant Biol 6:379389 Pignocchi C, Kiddle G, Hernndez I, Asensi A, Taybi T, Barnes JD, Foyer CH (2006) Apoplast redox state modulates gene transcription leading to modified hormone signalling and defence in tobacco

Administration Subcutaneous injection is the usual path