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You're most vulnerable during the initial titration period when your body is adapting to the medication's effects
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Future research can include randomized controlled trials and see if there are similar effects in other populations
Human studies of glucagon receptor antagonism show a striking result: hepatic amino-acid clearance drops hyperaminoacidaemia develops skeletal muscle becomes the fallback source of amino acids This pattern mirrors the very changes we see under GLP-1RA therapy
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