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A lower level of glutathione can contribute to a variety of adverse health effects

Two weeks at the lowest effective dose reveals individual sensitivity

Moreover, mitochondrial dysfunction and related oxidative stress can activate the innate branch of the immune system, leading to the release of pro-inflammatory cytokines influencing various depression-related pathophysiological mechanisms: monoaminergic neurotransmission disruption, tryptophan degradation toward neurotoxic catabolites, glutamate-related excitotoxicity, decreased neurotrophic factors and alterations in the hypothalamic-pituitary-adrenal axis [3, 33, 59]

TB-500 is a synthetic 17-amino-acid fragment corresponding to the actin-binding domain of thymosin beta-4 (T4), an intracellular protein involved in G-actin sequestration and cell migration regulation

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