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These findings lend support to the hypothesis that high levels of oxidative stress may contribute to the pathophysiology of depressionthough its effects may be anatomically selective
Evidence that TDP-43 is not the major ubiquitinated target within the pathological inclusions of amyotrophic lateral sclerosis
The Lancet (2023) 8
Activation of NOX enzymes in Kupffer and hepatic stellate cells induces ROS generation, thereby promoting liver fibrosis and hepatocarcinogenesis 93,94,95,96
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