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European Journal Of Heart Failure 2023, 25: 2274-2286
detected chemokines in a conditioned medium of AILI-derived hepatocytes revealing the higher abundance of VEGF-D (11-fold), VEGF (1.7-fold), its receptors VEGF-R3 and VEGF-R2 (sixfold), and lower expression of VEGF-R1 (0.7-fold) and HGF-R (0.6-fold), and VEGF may activate the STAT3 and ATM pathways[186]
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