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Its unique structure enables it to regain its active form after neutralizing free radicals, allowing it to continue protecting the body from oxidative damage

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The female mice (homozygous for Hba tm1(HBA)Tow and heterozygous for Hbb tm2(HBG1,HBB*)Tow and Hbb tm3(HBG1,HBB)Tow [h/h:: A / S ], sickle-cell trait mice) and male mice (homozygous for both Hba tm1(HBA)Tow and Hbb tm2(HBG1,HBB*)Tow [h/h:: S / S ], sickle SCD mice) were bred to generate SCD mice

Although that study's focus was not limited to vesicle ultrastructure, it links a mitochondrial quality control defect to detectable alterations in host immune response dynamics during infection (130)

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