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Szabo, another former Selye student, built on those findings to show that cytoprotective compounds work, in part, by protecting the blood vessels just beneath the stomach lining
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An 86% reduction in risk of new vertebral fractures
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This suggests that direct COP1-p53 recognition may require additional cofactors, adaptors, or specific cellular signal conditions such as glucose induce assembly of CRL4 COP1 for p53 degradation, as suggested in Su et al 29
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