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suggested that KPV may enter epithelial and immune cell cultures through the PepT1 transporter, where it appeared to slow IB- degradation and shorten NF-B activation windows while reducing ERK1/2, JNK, and p38 phosphorylation and lowering IL-8 output in these experimental settings
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The tripeptide workspeer-reviewed studies confirm NF-B inhibition and measurable reductions in TNF-, IL-6, and IL-1 in validated models
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