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GLP-1 addresses all five risk factors simultaneously with a single weekly injection
It does not replace medical advice, diagnosis, or treatment
3 The findings were significant: Semaglutide improved both cognitive and motor function in mice with MS-like disease Reduced demyelination (the nerve damage that characterises MS) Promoted remyelination (nerve repair) Reduced neuroinflammation by lowering inflammatory markers Protected against oxidative stress damage This was the first study to examine semaglutide specifically in an MS model, and the results suggest that the medication may have direct neuroprotective effects beyond its weight-loss benefits
Downstream production of pro-inflammatory cytokines such as interleukin-4 (IL-4), interleukin-5 (IL-5), and interleukin-13 (IL-13) contribute to chronic inflammation and fibrosis ( + lymphocytes (Liberman et al., 2013)
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Mechanisms of GLP-1 receptor agonists: An overview
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