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Excessive production of ROS, largely arising from mitochondrial oxidative phosphorylation, is a major driver of oxidative damage and mitochondrial dysfunction processes that correlate with MDD symptom severity, illness chronicity, cognitive impairment, and poor antidepressant response [138, 139]
All of these non-radical oxidants can induce protein modifications and mtDNA damage
KEGG pathway enrichment analysis was conducted to identify the potential signaling pathways associated with potential targets, and the analysis uncovered several significant pathways 15 (Fig
Best used together, not against each other
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