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GHK-Cu displaces Keap1s Cu coordination (Log K ~16 for GHK-Cu versus ~8 for Keap1 cysteine-zinc coordination) to stabilise Nrf2 and drive nuclear translocation
[13] [14] Clinical trials have shown reductions in major adverse cardiovascular events, including myocardial infarction and stroke, with several agents in this class
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People with a history of cancer should be particularly wary, as IGF-1 (which increases with CJC-1295) may be involved in the development of certain types of cancer
Why is GHK-Cu dosed at 50mg while the other components are 10mg each
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