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The research rationale for a selective NNMT inhibitor is to interrupt exactly that diversion, so the cell keeps more of its NAD+ pool and can run its mitochondrial machinery again

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The malignancy caution is not incidental: because GH and its downstream mediator IGF-1 can promote cell growth, any setting where cancer may be present is a firm contraindication in Tesamorelins own label, and that logic carries directly to the blend

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