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[] This study also discovered that heavy metals are able to deactivate GSH through attachment to the sulfhydryl group, thereby causing ineffective GSH replenishment and therefore creating oxidative stress

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However, it is encouraging that preclinical and early human studies suggest that GLP-1 agonism appears broadly protective: indeed clinical trials of these agents are being initiated in cardiovascular disease (SELECT and SOUL trials, see below), NASH, and neurodegeneration (Alzheimers and Parkinsons)

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