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A 2011 mechanism paper by Chang and colleagues in Journal of Applied Physiology demonstrated that BPC-157 promotes tendon fibroblast outgrowth, supports fibroblast survival under oxidative stress, and facilitates cell migration through the FAK-paxillin signaling pathway a pathway central to cell adhesion and movement

Similarly, patients with active malignancy warrant careful consideration given theoretical concerns about angiogenic effects potentially affecting tumor growth, though preclinical evidence suggests context-dependent effects that may inhibit rather than promote tumor angiogenesis

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